Search results for "Microbial Collagenase"

showing 3 items of 3 documents

Ultrastructural evidence of collagenolytic activity in ductal infiltrating carcinoma of the human breast

1987

The stroma of ductal infiltrating carcinoma of the human breast shows characteristic and localized areas of collagen rarefaction and fragmentation. This finding has been correlated with a peculiar type of fibrillar damage, observed in a small percentage of collagen fibrils isolated in the native state from the tumour stroma. The same pattern of lesion has been reproduced in vitro by human collagenase digestion on reconstituted fibrils. No effect has been detected by other nonspecific proteases in the same system.

Cancer ResearchProteasesPathologymedicine.medical_specialtyMammary glandBreast Neoplasmsmacromolecular substancesBiologyLesionStromamedicineHumansTrypsinFragmentation (cell biology)AgedPancreatic ElastaseMiddle AgedIn vitroMicroscopy ElectronCarcinoma Intraductal NoninfiltratingMicrobial Collagenasemedicine.anatomical_structureOncologyCollagenaseUltrastructureFemaleCollagenmedicine.symptommedicine.drug
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Enzymatic alteration of C1q, the collagen-like subcomponent of the first component of complement, leads to cross-reactivity with type II collagen

1988

AbstractNative serum C1q, the collagenous-like subcomponent of the first component of complement, is not recognized by polyclonal anti-collagen type II antibodies. However, when purified C1q was subjected to limited proteolysis by collagenase it showed antigenic cross-reactivity with collagen type II. The same cross-reactivity was observed with hemolytically active C1q in synovial fluids of patients with rheumatoid arthritis (RA), whereas C1q from synovial fluids of patients with osteoarthritis (OA), villo-nodular synovitis and ankylosing spondylitis was not recognized by this antibody. However, incubation of synovial fluid C1q of OA patients with synovial fluid leucocytes from RA patients …

Complement Activating EnzymesCollagenaseComplementBiophysicsType II collagenEnzyme-Linked Immunosorbent Assaychemical and pharmacologic phenomenaOsteoarthritisBiochemistryAntibodiesArthritis Rheumatoidfluids and secretionsAntigenComplement C1immune system diseasesStructural BiologySynovitisOsteoarthritisSynovial FluidGeneticsmedicineAnimalsHumansSynovial fluidSpondylitis AnkylosingAntigensRheumatoid arthritisskin and connective tissue diseasesMolecular BiologyC1qAutoantibodiesSheepSynovitisbiologyChemistryComplement C1qAntibodies MonoclonalCell Biologymedicine.diseaseMolecular biologyMicrobial CollagenasePolyclonal antibodiesImmunologyCollagenasebiology.proteinCollagenAntibodyGranulocytesmedicine.drugFEBS Letters
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Activation of complement by the alternative pathway as a factor in the pathogenesis of periodontal disease.

1976

Dental plaque and a bacterium, Actinomyces viscosus, isolated from plaque that can reproduce periodontal disease in germ-free rats, are activators of complement by the alternative pathway. It is suggested that this process is involved in the pathogenesis of chronic inflammatory periodontal disease.

T-LymphocytesGuinea PigsDental PlaqueAntigen-Antibody ComplexDental plaquePathogenesisstomatognathic systemPeriodontal diseasemedicineActinomycesAnimalsHumansActinomyces viscosusBone ResorptionPeriodontitisGlycoproteinsB-LymphocytesEnzyme Precursorsbusiness.industryMacrophagesGlobulinsGeneral MedicineComplement C3Complement System Proteinsmedicine.diseaseCathepsinsComplement (complexity)RatsEndotoxinsstomatognathic diseasesMicrobial CollagenaseImmunologyAlternative complement pathwaybusinessLancet (London, England)
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